NBIDPSTM

Neural basis for individual differences in pSTM in the normal and dyslexic populations

 Coordinatore MEDICAL RESEARCH COUNCIL 

 Organization address address: NORTH STAR AVENUE POLARIS HOUSE
city: SWINDON
postcode: SN2 1FL

contact info
Titolo: Ms.
Nome: Anthea
Cognome: Hills
Email: send email
Telefono: +44 1223 355 294
Fax: +44 1223 516 889

 Nazionalità Coordinatore United Kingdom [UK]
 Totale costo 0 €
 EC contributo 171˙867 €
 Programma FP7-PEOPLE
Specific programme "People" implementing the Seventh Framework Programme of the European Community for research, technological development and demonstration activities (2007 to 2013)
 Code Call FP7-PEOPLE-IEF-2008
 Funding Scheme MC-IEF
 Anno di inizio 2009
 Periodo (anno-mese-giorno) 2009-03-02   -   2011-03-01

 Partecipanti

# participant  country  role  EC contrib. [€] 
1    MEDICAL RESEARCH COUNCIL

 Organization address address: NORTH STAR AVENUE POLARIS HOUSE
city: SWINDON
postcode: SN2 1FL

contact info
Titolo: Ms.
Nome: Anthea
Cognome: Hills
Email: send email
Telefono: +44 1223 355 294
Fax: +44 1223 516 889

UK (SWINDON) coordinator 171˙867.62

Mappa


 Word cloud

Esplora la "nuvola delle parole (Word Cloud) per avere un'idea di massima del progetto.

cognitive    pstm    theoretical    questions    upon    literacy    deficit    developmental    substrate    phonological    memory    dd    central    deficits    representation    disorder    alternative    clinical    dyslexia    hereditary    neurodevelopmental    neural   

 Obiettivo del progetto (Objective)

'Developmental dyslexia (DD) is a hereditary neurodevelopmental disorder, affecting 5%-10% of the population. It is associated with impoverished literacy skills despite normal intelligence and adequate educational opportunities, and occurs in the absence of major sensory impairments. There is currently much interest in the proposal that dyslexia is best characterised as a language-specific deficit in which literacy problems stem from impairment in the representation and processing of phonemes. However, a growing body of empirical evidence fails to support this view. The research proposed here tests an alternative theoretical stance, namely that deficits in phonological short-term memory (pSTM) are central to the literacy problems observed in DD. Within the fields of cognitive psychology and cognitive neuroscience, there have been attempts to further understand the functions and the neural substrate of pSTM. However, fundamental questions concerning the role of pSTM in speech processing remain unanswered and little attempt have been made to translate these basic cognitive science studies to address vital clinical questions, specifically the centrality of deficits in pSTM as opposed to phonological representation and processes in DD. Consequently, the aim of the proposed research is twofold: First, we need to more clearly understand the relationship between phonological processing and pSTM processes and the extent to which they rely upon common or distinct neural substrates before we can establish where the deficit lies in DD. Hence the core components of this proposal focus on this initial step, where we aim to investigate the intersection of pSTM and phonological processing by assessing their overlapping neural networks. Secondly, we aim to extend this and make a step towards translating the results into clinical research addressing the question whether dyslexics’ deficit is more related to one or other, or common substrate impacts upon both.'

Introduzione (Teaser)

Researchers taking an alternative theoretical approach to developmental dyslexia (DD) hope to shed new light into this hereditary neurodevelopmental disorder. They studied whether deficits in phonological short-term memory (pSTM) are central to literacy problems in DD.

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