BRYOZOA

Ecological genetics of Bryozoa-Myxozoa host-parasite interactions

Coordinatore	EIDGENOESSISCHE ANSTALT FUR WASSERVERSORGUNG ABWASSERREINIGUNG UND GEWAESSERSCHUTZ    more  Organization address address: UEBERLANDSTRASSE 133 city: DUEBENDORF postcode: 8600 contact info Titolo: Ms. Nome: Laura Cognome: Klein Email: send email Telefono: +44 823 5388 Fax: +41 44 823 5818
Nazionalità Coordinatore	Switzerland [CH]
Totale costo	109˙290 €
EC contributo	109˙290 €
Programma	FP7-PEOPLE Specific programme "People" implementing the Seventh Framework Programme of the European Community for research, technological development and demonstration activities (2007 to 2013)
Code Call	FP7-PEOPLE-2009-IEF
Funding Scheme	MC-IEF
Anno di inizio	2010
Periodo (anno-mese-giorno)	2010-05-01   -   2011-07-31

 Partecipanti

#	participant	country	role	EC contrib. [€]
1	EIDGENOESSISCHE ANSTALT FUR WASSERVERSORGUNG ABWASSERREINIGUNG UND GEWAESSERSCHUTZ  Organization address address: UEBERLANDSTRASSE 133 city: DUEBENDORF postcode: 8600 contact info Titolo: Ms. Nome: Laura Cognome: Klein Email: send email Telefono: +44 823 5388 Fax: +41 44 823 5818	CH (DUEBENDORF)	coordinator	109˙290.75

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 Obiettivo del progetto (Objective)

'Understanding the clonal and population genetic structure of the freshwater bryozoan Fredericella sultana is essential for understanding the epidemiology and management of the myxozoan parasite Tetracapsuloides bryosalmonae that causes the Proliferative Kidney Disease (PKD) in wild and farmed salmonids. Common, widespread and susceptible F. sultana clones can serve as a persistent parasite reservoir, supplying disease epidemics in fish, yet, at present, no information of the clonal structure of F. sultana populations exists. To predict the impact of environmental change on spread of PKD in fish, it would be essential to resolve questions regarding geographic distribution of susceptible and resistant F. sultana genotypes, and their potential response to environmental change. At present such information is not available due to lack of neutral genetic markers for F. sultana. This 15 month fellowship proposal will (i) develop the required nuclear genetic markers that will allow high resolution genetic studies of the clonal and population genetic structure of F. sultana populations, and (ii) use these markers in the first genetic surveys of F. sultana populations that are either known to carry the disease or known to be healthy, based on earlier ecological surveys. Results of the study will be highly beneficial for preventive and predictive applications to manage PKD in salmonids.'